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In one study on the Shank3-deficient ASD mouse model, the chronic administration of OXT prevented social deficits after 2 weeks of treatment but led to social ambivalence beyond that

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In the H 2 O 2 -induced melanocyte model, Hyp protected melanocytes from oxidative damage by regulating the PI3K/Akt pathway, inhibiting p38 phosphorylation and suppressing mitochondrial apoptotic signaling, which included upregulation of the Bcl-2/Bax ratio and expression of Akt, and downregulation of caspase 3, p38 (Yang et al., 2016)

These nutrients work together to help convert food into fuel, support brain health, and optimize metabolic performance