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The release of free copper ions, facilitated by transporters such as ATP7A and ATOX1, induces neointimal thickening following vascular injury, contributing directly to lesion development [193, 194]

Thymosin 4s core molecular function is well established: it sequesters monomeric G-actin, binding it roughly 1:1 and regulating the balance between free actin and the polymerized filaments that drive cell shape and movement

A MTHFR mutation can be heterozygous (1 abnormal gene) or homozygous (2 abnormal genes)

in 2018, exhibits an IC 50 value of 1 M for CBS and shows approximately 30-fold higher inhibitory activity toward CBS than toward CSE (Figure 4B) (Wang et al., 2018)