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The muscular oxidative-stress-mediated inflammation in myoblast cells can be abrogated by the AhR inhibitor CH-223191, the antioxidant ascorbic acid, or the OAT inhibitor probenecid

WT endometrium-recipient mice fed HFD (45% kcal from fat) showed reduced lesion incidence, numbers, and volumes, in the absence of changes in systemic ovarian steroid hormone and insulin levels, relative to those fed the control diet (CD, 17% kcal from fat)

However, it is not yet clear how autoimmune responses of GD are activated

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